Preliminary experiments suggest conolidine could inhibit unique ion channels, minimizing neuronal excitability and limiting pain signals. This mechanism is particularly pertinent in neuropathic suffering, where by abnormal signaling leads to persistent discomfort. Furthermore, conolidine appears to affect G protein-coupled receptor (GPCR) pathways integral to agony perception. Computa... https://hamidm937omk7.wikisona.com/user