These reports recognize intersecting roles for different ALS-involved proteins, implicating ATXN2 in complex and probably pathological cascades. These results also reveal that ATXN2 impacts several sickness-associated processes such as autophagy, apoptosis and mRNP formation. It continues to be unclear if FUS mutations result in a loss of functionality in the https://billw000iue3.blog-gold.com/profile